Application: | Internal Medicine |
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Usage Mode: | Injection |
Suitable for: | Elderly, Children, Adult |
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Basic Info.
Pharmaceutical Technology
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Chemical Synthesis | Function | Improving myocardial function |
Specification | 1g | Standard Available | BP, CP, USP |
OEM/ODM | Support | Origin | China |
Delivery Time | 30~60 Days | Transport Package | Carton |
HS Code | 300490909 | Production Capacity | 1,000,000Boxes/Month |
Creatine Phosphate Sodium for Injection
Indications:
Creatine Sodium phosphate is a non cardiac glycoside cardiovascular system drug, mainly used clinically to protect the myocardium and improve myocardial metabolic abnormalities during surgery.
Protecting the myocardium: Adding this product to cardiac arrest fluid during cardiac surgery can protect the myocardium.
Improving myocardial metabolic abnormalities: Used to improve myocardial metabolic abnormalities under ischemic conditions, including myocardial damage caused by pneumonia combined with heart failure, neonatal pathological jaundice, neonatal asphyxia and hypoxia, neonatal hypoxic-ischemic encephalopathy, viral myocarditis, enterovirus infection, etc; Congenital heart disease, myocarditis, heart failure, etc.
Phosphate creatine plays an important role in the energy metabolism of muscle contraction. It is the chemical energy reserve of the myocardium and skeletal muscles, and can be used for ATP biosynthesis. The hydrolysis of ATP provides energy for the contraction process of actin.
The insufficient energy supply caused by slowed oxidative metabolism is an important factor in the formation and development of myocardial cell damage. Insufficient levels of creatine phosphate are common in the impairment of myocardial contractility and functional recovery ability. In fact, there is a close relationship between the amount of high-energy phosphate compounds in cells and their ability to survive and recover contractile function during myocardial damage. High energy phosphate compounds can protect the myocardium from damage and also have a protective effect on myocardial metabolism.